Michele Evans Wikipedia, Md, Fetility, Facebook, Salary, Linkedin

Michele Evans Wikipedia, Md, Fetility, Facebook, Salary, Linkedin

Michele Evans Wikipedia, Md, Fetility, Facebook, Salary, Linkedin – Internist and medical oncologist Dr. Michele K. Evans earned her undergraduate degree from Barnard College at Columbia University and her medical degree from the Robert Wood Johnson Medical School at Rutgers, The State University of New Jersey, in Piscataway.

Michele Evans Wikipedia, Md, Fetility, Facebook, Salary, Linkedin

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Michele Evans Wikipedia, Md, Fetility, Facebook, Salary, Linkedin

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Michele Evans Wikipedia, Md, Fetility, Facebook, Salary, Linkedin

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Michele Evans Wikipedia, Md, Fetility, Facebook, Salary, Linkedin

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She completed post-graduate study in internal medicine at Emory University School of Medicine and a fellowship in medical oncology at the National Cancer Institute (NCI)’s Medicine Branch of the Clinical Oncology Programme. Dr. Evans oversees training for the NIA Intramural Research program and is the NIA’s deputy scientific director. She has a background in medical science and does basic bench research on the biology of health disparities and minority aging in addition to epidemiologic clinical research in health disparities.

At this moment, it is unresolved what biological and molecular processes affect people’s health and lifespan trajectories in American culture. We perform integrated clinical and fundamental science research with the goal of identifying the underlying factors contributing to the disproportionate incidence, morbidity, and death of age-related illnesses among minorities and Americans with low socioeconomic status (SES).

The strategy offers a two-way link between clinical research and basic science laboratory studies that extends from the targeted epidemiologic population to the bench. Understanding how race, socioeconomic status, culture, behavior, exposure to the environment, biological vulnerabilities, genetics, social environment, access to health care, and quality of care interact to produce unjust rates of age-related disease and disability is possible.

It is thought that DNA repair processes are essential for maintaining the integrity of the genome. Tumorigenesis is aided by genetic lesions building up, a loss of fidelity in the replicative machinery, and insufficient DNA repair systems. Similar to this, aging or cellular senescence is defined by the random buildup of DNA, RNA, or protein damage or mutation, as well as possible declines in DNA repair capacity. Age-related increases in cancer incidence provide more evidence of the molecular similarity between these two biological processes.

The impaired ability to repair DNA appears to be the critical and convergent component underlining the important clinical symptoms associated with DNA repair pathway abnormalities. The overarching goal of our research has been to learn more about the role of DNA repair in cellular senescence and carcinogenesis in order to find strategies to employ measured DNA repair capacity as a clinical tool in the detection and treatment of cancer, as well as age-related disease and disability.

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